Carotid endarterectomy (CEA)

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UNDER CONSTRUCTION

Anatomy Review

Typical anatomy: ICA is posterior-laterally while ECA is anterior-medial (as in diagram below)
  • but often this is rotated such that ICA is abutting tracheo-esophageal space → “kissing carotids”
 
Common Carotid branches
  • superior thyroid artery
  • ascending pharyngeal
    • very relevant surgically
Carotid sheath:
  • deep to SCM, in front of longus colli
ECA:
  • branches either coming off posteriorly or anteriorly
 
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Booking

  • awake vs. asleep
  • neurophys vs. TCDs

Trivia

  • what is the largest muscle in a horse? Platysma
 

Incision

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Exposure:

  • SCM takes you to carotid sheath. work along anterior border, mobilize laterally to come on the carotid sheath
  • Common facial vein: generally found at carotid bifurcation
    • not always present, can also be duplicated, truplicated into smaller branches
    • generally speaking, will ligate it to mobilize the jugular vein
  • Facial artery: found about 1-2 cm anterior to angle of mandible (you can palpate on your own head)
    • marginal mandibular nerve runs right under this
      • fixed retraction → marginal mandibular palsy (lower lip muscle weakness) → patients cannot drink!
  • Vagus nerve:
    • the third structure in carotid sheath
    • generally deep to IJV (lateral) and deep to ICA (medial) however can also be superficial
  • Hypoglossal nerve:
    • muscular landmark: posterior belly of digastric muscle
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Occluding carotid

ICE (internal, common, external)
  • heavily calcified plaques thin out wall, do not punch through as you’re creating plane!
 
 

Optional: shunting

  • generally do not need this if clamp time < 20 minutes or so
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Closure

(LEFT) primary closure. (RIGHT) patch angioplasty. note that this increases diamater at level of stenosis which may decrease restenosis risk logically however also may increase it by disrupting laminar flow, increasing clotting risk.
(LEFT) primary closure. (RIGHT) patch angioplasty. note that this increases diamater at level of stenosis which may decrease restenosis risk logically however also may increase it by disrupting laminar flow, increasing clotting risk.